Dietary Components For The Diabetes Treatment

Base your diet recommendations on treatment goals designed specifically for your patient . Consider the various roles of proteins, carbohydrates, fats, sweeteners, fiber, sodium, and alcohol in your patient’s diet.

Cultural and Ethnic Considerations

Each patient’s cultural and ethnic background strongly influences her food customs, eating rituals, food preparation, and body image. Religion also can affect dietary habits. For example, Hindus are vegetarians, and Orthodox Jews follow kosher dietary laws.

Family traditions may dictate mealtime habits and foods to be eaten or avoided. A patient’s finances, social status, and geographic region affect the type and availability of foods she eats, as well. The health care team performs a thorough nutritional assessment of cultural and ethnic practices and incorporates them into a personalized nutritional plan.

Food Labels

The Food and Drug Administration’s requirements for food labels have made a big difference to patients with diabetes as they shop for food. The information on labels is useful not only for assessing individual products but also for comparing ingredients of similar products and of different brands of the same product.

Show your patient several labels of healthful and less healthful foods. Point out that many imported foods lack nutritional information.

Ingredients on food labels are listed in descending order by weight. Determining total sugar content may take some analysis, however, because different forms can be listed separately. Give your patient a list of sugar’s many names, including sorghum, sucrose, lactose, and maple syrup. Explain that foods labeled dietetic aren’t necessarily sugar-free and that natural doesn’t mean sugar-free. Cane sugar, for example, is natural. Dietetic foods are usually more expensive, and they’re unnecessary for patients who make an effort to choose foods intelligently.

Food labels also list the number of calories, total fat content, and amount of saturated fat per serving. The difference between total fat and saturated fat is the portion that consists of polyunsaturated or monounsaturated fats. The polyunsaturated and monounsaturated fat content should be greater than the saturated fat content. Also listed are levels of cholesterol, sodium, total carbohydrate, fiber, sugar, and protein.

Dietary Components For The Diabetes Treatment

A food label promising no cholesterol can be misleading. Vegetable oils containing no cholesterol, for example, may be high in saturated fats. Also, teach your patient to scrutinize labels claiming that a food is a certain percentage fat free. A product that is 75% fat free contains 25% fat by weight-and even more than 25% of the total calories may come from fat. The total calories from fat, listed next to the total calories on the label, will give your patient a clearer picture of fat content. Teach her to carefully read labels boasting fewer calories, light, or lite.

Besides ingredients, food labels now list details of nutritional content. This information makes it easier for your patient to choose foods in accordance with her treatment goals.

The information on the food label is based on the serving size, which appears at the top of the food label. Make sure your patient understands that the nutritional content of the entire package isn’t being described unless the label states that the container has only one serving. The percent of daily value listed on the label is based on a 2,000-calorie diet; teach patients with a different calorie plan to take this into consideration.

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Coronary Artery Disease or CAD

The most common cause of death in patients with Type 2 diabetes, CAD also develops in many patients with Type 1 diabetes. Patients who have had diabetes for 20 years or longer, are over age 40, or have many cardiovascular risk factors, such as hypertension, obesity, or lipid abnormalities, have a high risk of developing CAD. Men and women with diabetes develop CAD at about the same age.The development of atherosclerotic changes in the coronary arteries is closely related to the duration and severity of diabetes. The prolonged high levels of blood glucose, free fatty acids, and cholesterol damage the endothelial layer of the arteries. Monocytes adhere to the damaged arteries, and macrophages migrate to these areas. If your patient also has hypertension, the high pressure of blood flowing through the vessels increases the endothelial damage.

This all contributes to lipid accumulation and the development of atherosclerotic plaque. The macro phages release growth factor, which stimulates smooth-muscle cells to enlarge, multiply, and migrate through the layers of the arteries. This further narrows the lumen of the arteries. At the same time, increased numbers of platelets adhere to the damaged endothelium, which causes thrombus formation. If your patient has hyperglycemia, platelets are more likely to adhere to the artery wall. The combination of endothelial damage and increased platelet aggregation leads to accelerated thickening of the lumen, which results in vasoconstriction.

If your patient has insulin resistance, her triglyceride and lipoprotein levels will be elevated, which can damage the endothelial lining even further. Her pancreas produces excessive amounts of substandard insulin to overcome the body’s resistance to insulin. So, her body does not have sufficient, effective insulin, which is required to produce lipoprotein lipase, the enzyme that regulates cholesterol.

Coronary Artery Disease or CAD Progressive atherosclerosis, of course, reduces coronary artery blood flow, which increases the risk of developing myocardial ischemia and infarction.

If your patient has a myocardial infarction (MI), she’s at risk for developing diabetic ketoacidosis (DKA). The stress of an Ml causes the release of adrenal corticosteroids and catecholamines, which inhibit insulin action and stimulate glucose production. As the glucose level rises, the heart and other vital organs are deprived of their main energy source. As a protective mechanism, the body begins to break down fat to use as a substitute energy source. Fat breakdown is incomplete, causing excessive amounts of ketones to be released into the bloodstream. Because the kidneys can’t adequately excrete the ketones, DKA develops. This condition results in electrolyte disturbances, which can lead to life-threatening cardiac arrhythmias.

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Benefits of Carbohydrates in Diabetes

Carbohydrates supply the body’s primary source of energy. The brain and red blood cells use only glucose, the building block of carbohydrates. The American Diabetes Association recommends determining your patient’s protein requirements, using the recommended dietary allowance of 0.8 g/kg of body weight for adults, before determining the desired amount of carbohydrates and fat. Also, use your patient’s treatment goals, habits, and blood glucose and lipid goals as guides.When your patient chooses which carbohydrates to consume, instruct her to focus on the glycemic value of a carbohydrate rather than on the type of carbohydrate, such as simple or complex. The glycemic index, created in 1981, provides average glycemic values of certain foods. It can help you predict the rise in blood glucose after your patient has eaten certain carbohydrates. Foods that raise blood glucose levels quickly have a high glycemic value. These include white bread, some cereals, glucose, and root vegetables, such as carrots and potatoes. Foods with a low glycemic value include nuts, legumes, dairy products, fructose, and raw fruits.

Advise your patient to use the index as a guide and to monitor her blood glucose levels after she eats certain foods. Make it clear, however, that the glycemic value of a food is not equivalent to its nutritional value. Although sucrose has a lower glycemic value than a potato, for example, it provides far less nutritional value. And inform your patient that the glycemic value of a food can rise or fall depending on many factors, including the ripeness of the food, the preparation of the food, and the other foods eaten at the same meal.

Benefits of CarbohydratesIf your patient is using regular insulin, she can count grams of carbohydrates to help her make food choices. Instruct her to count the total number of grams of carbohydrate in a meal she’s planning to eat. She should then compare that number to the amount of carbohydrate recommended for that particular meal in her meal plan. If she’s consuming more than is recommended, she can increase her regular insulin dose by 1 unit for every additional 10 grams of carbohydrate. If she’s consuming fewer carbohydrates than is recommended, she can decrease her insulin by 1 unit for every 10 grams less.

Instruct your patient to count grams of carbohydrates by consulting food labels, exchange lists, and carbohydrate-counting books. Counting grams of carbohydrate allows for more accurate insulin dosing and more flexible meal scheduling. It also reduces the number of hypoglycemic and hyperglycemic episodes.

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Facts About Hypoglycemia

Whether your patient recently learned that she has diabetes or she has been treating herself for years, during her hospitalization you should teachor reinforce-certain essential information about hypoglycemia. Be sure you cover the following areas:

  • Explore the possible causes of hypoglycemia.
  • Discuss the signs and symptoms of hypoglycemia.
  • Discuss how to recognize and treat hypoglycemia promptly.
  • Remind her to carry a simple carbohydrate snack, such as peanut butter crackers, at all times to treat hypoglycemia.
  • Explain the importance of wearing a bracelet or necklace or carrying a card that indicates she has diabetes.Facts About Hypoglycemia
  • Discuss the importance of telling family and friends about her diabetes in case she has a hypoglycemic episode.
  • If your patient uses insulin, recommend that she obtain a glucagon kit and teach her family and friends when and how to use it.
  • Teach her to monitor her blood glucose level frequently.
  • Explain the importance of a regular pattern for eating meals and administering insulin or oral antidiabetic drugs.
  • Explain the need to adjust the amount of food, the timing of meals and snacks, or the times of insulin administration when she engages in strenuous physical activity.
  • Explain the importance of checking with the physician before taking a new prescription or over-the-counter drug.
  • Instruct her to tell her health care providers that she has diabetes and that she uses insulin or oral anti diabetic drugs, as appropriate.

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Nursing Considerations after Kidney and Pancreas Transplant

When caring for a patient after a kidney and pancreas transplant, repeatedly assess the function of both organs. Assess your patient for hypovolemia and dehydration, which may damage the kidney tubules and increase the risk of thrombosis of the pancreatic vessels. Monitor the patient’s intake and out­put, vital signs, skin turgor, blood urea nitrogen (BUN) level, creatinine level, and hematocrit. Elevated BUN and creatinine levels may indicate kidney dysfunction and rejection. A steadily declining hematocrit may reflect bleeding.

For the first 24 hours after surgery, adjust I.V. fluids every hour in response to your patient’s urine and nasogastric (NG) output. If she has an indwelling urinary catheter, you can monitor urine output accurately. The catheter also decompresses the bladder, allowing the suture line within to heal. After the NG tube and urinary catheter are removed, continue to monitor your patient’s intake and output.

Monitor blood glucose levels every 30 minutes to 1 hour for the first 24 hours after surgery and administer insulin or fluids containing dextrose as necessary. A patient may require insulin for 1 to 2 days after surgery because the function of the transplanted pancreas may be delayed. She may also require insulin if she’s receiving steroid therapy.

Nursing Considerations after Kidney and Pancreas TransplantReview your patient’s daily laboratory test results, including electrolytes, complete blood count, hemoglobin level, and serum and urine amylase measurements. Collect urine specimens for cumulative amylase determinations taken at 12-hour or 24-hour intervals. Assess insulin, glucagon, and human C peptide levels three or four times per week. Pancreas scans and ultrasound examinations may be performed on the first day after surgery, then once a week until discharge. Ultrasounds examine the function of the new organs and enable the physician to determine the size of the graft and the patency of the veins.

Check your diabetes patient’s abdominal dressing and incision for blood and urinary drainage and for signs of infection. To decrease postoperative complications, have your patient sit on the side of her bed within 24 hours of surgery. She can begin sitting in a chair and taking short walks the day after surgery. Encourage her to cough, breathe deeply, and change positions frequently.

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Curing method of Diabetic Retinopathy

The treatment of diabetic retinopathy depends on the extent of retinal damage and may include laser photocoagulation or vitrectomy. With laser photocoagulation, the ophthalmologist uses laser beams to seal microaneurysms stream. The vascular lens refracts and focuses images onto the retina.

The choroid, or middle coat, is made up of many arteries and veins. The retina, the innermost coat of the eyeball, is rich in neurons, including the rods and cones, which serve as visual receptors. The retina is connected to the optic nerve, which conducts visual information to the brain. The vitreous humor-a thick, gelatinous material-fills the space behind the lens. It maintains the shape of the eye­ball and placement of the retina. and thus reduce their risk of bleeding. Laser photocoagulation can also be used to control new blood vessel growth .

The ophthalmologist uses vitrectomy, a surgical procedure, to treat advanced complications of proliferative retinopathy, such as vitreous hemorrhage and tractional retinal detachment.

Curing method of Diabetic RetinopathyVitrectomy requires the use of local or general anesthesia. To begin the procedure, the ophthalmologist makes a small incision behind the cornea. Then he removes blood and fibrous tissue from the vitreous humor and replaces them with another fluid, such as normal saline. A silicone oil or gas can be used to hold the retina in place.

Riskier than laser photocoagulation, vitrectomy can result in total vision loss. Because of this, vitrectomy is typically performed on patients with a high risk of complete vision loss – for example, those with bleeding into the vitreous humor and some vision loss that hasn’t resolved after 6 months.икони

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Treatment of Peripheral Neuropathy

If your patient maintains better blood glucose control, her pain and other symptoms may decrease. As nerve cells regenerate with improved blood glucose levels, your patient’s pain may worsen initially, but it will decrease over time.To prevent foot complications, your patient may need to use lamb’s wool padding to protect her feet from trauma. The physician may also refer her to a podiatrist, who will assess her feet regularly and gently file any callused areas, if necessary. Your patient may also need a referral to an orthotic or other foot care specialist to fit her for custommade shoes, molded insoles, or other orthotic devices to protect her feet. In some cases, your patient may require bed rest or crutches. Some patients with foot ulcers have casts applied so that they can walk while the ulcers heal. The cast redistributes foot pressure so that the ulcerated area bears much less weight than it would normally.

For pain associated with peripheral neuropathy, the physician may prescribe:

  • a nonnarcotic analgesic, such as ibuprofen or sulindac
  • tramadol hydrochloride
  • phenytoin or carbamazepine
  • a tricyclic antidepressant, such as amitriptyline, alone or with a phenothiazine derivative
  • mexiletine
  • topical capsaicin 0.075%.

Narcotic analgesics usually aren’t prescribed because peripheral neuropathy is a chronic condition, and the patient would risk developing an addiction. However, other therapeutic options may include transcutaneous electrical nerve stimulation therapy or referral to a pain control clinic.

Curing Steps for Patients

If your patient experiences painful paresthesia, especially at night, tell her to stretch gently or to get up and walk around. Also, teach her how to safely massage her feet and legs, and explain that this sometimes helps to block pain sensations. If appropriate, teach your patient how to safely use the TENS unit. Teach her about guided imagery, meditation, progressive relaxation techniques, exercise, and therapeutic massage.

Teach your patient the importance of foot care, and review safety measures she can take to prevent trauma. For instance, advise her to avoid prolonged standing and strenuous weight-bearing exercises. Tell her to wear well-cushioned, properly fitting shoes and to walk in well-lit areas where the ground is smooth.

If your patient has decreased temperature sensation, advise her to prevent severe burns by checking the water temperature with a part of her body that’s sensitive to temperature, such as forearm. If she experiences abnormal cold sensations, advise her to wear thin gloves in the spring or fall. When temperatures drop, she should wear heavier, insulated gloves. Tell her that mittens may be warmer than gloves. If cold feet are a problem, tell her to wear comfortable insulated socks with her shoes.

Treatment of Peripheral NeuropathyReview with your patient the name, dosage, action, and adverse effects of all her prescribed drugs. If she’s using topical capsaicin, tell her to wear gloves when she applies it, avoid contact with her eyes, and wash her hands immediately after application. Also, warn her that she may experience transient burning of the affected area after applying capsaicin.

Encourage your patient to avoid drinking alcohol. Explain that alcohol abuse may contribute to the progression of peripheral neuropathy. If appropriate, refer your patient to a counselor or to Alcoholics anonymous. If she smokes, encourage her to stop. Explain that cigarette smoking can worsen her condition and that stopping may slow its progress.

Explain that your patient should have her feet inspected at least four times a year and that a physician should perform a thorough physical examination and neurologic assessment at least once a year.

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Autonomic and Neuroglycopenic Signs and Symptoms

The early signs and symptoms of hypoglycemia tend to be autonomic. Neuroglycopenic signs and symptoms generally appear later.

Autonomic Signs and Symptoms

  • nervousness
  • tremors
  • palpitations
  • diaphoresis
  • anxiety
  • pallor
  • irritability
  • hunger
  • paresthesia

Autonomic and Neuroglycopenic Signs and Symptoms

Neuroglycopenic Signs and Symptoms

  • dizziness
  • headache
  • lack of coordination
  • difficulty concentrating
  • mental dullness
  • severe lethargy
  • slurred speech
  • blurred vision
  • mood changes
  • seizures
  • coma

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Acarbose- Glucosidase Inhibitor

Acarbose, an Intestinal Alpha-Glucosidase Inhibitor, decreases postprandial hyperglycemia by inhibiting the digestion and absorption of carbohydrates. It achieves this by inhibiting the enzymes responsible for the digestion of starches and other carbohydrates in the brush border of the small intestine.The peak action of acarbose occurs within 1 hour of ingestion. The drug is metabolized by intestinal bacteria and digestive enzymes. Because the half-life for acarbose is only about 2 hours, the drug should be taken three times a day.

A physician will begin acarbose therapy at 25 mg three times a day. She will adjust dosages every 4 to 8 weeks based on 1-hour postprandial blood glucose levels and on tolerance until an effective dose is achieved. The maximum recommended dosage is 50 mg three times a day for patients who weigh 60 kg or less. For those who weigh more than 60 kg, the maximum dosage is 100 mg three times a day.

Indications and ContraindicationsAcarbose Glucosidase Inhibitor

Acarbose is used with stable Type 2 patients as an adjunct to diet and exercise to reduce blood glucose levels. This drug benefits patients who can’t achieve near-normal blood glucose levels with sulfonylureas or metformin.

Because acarbose increases gas formation in the intestine, it’s contraindicated for anyone with inflammatory bowel disease, ulcerations of the colon, or intestinal obstruction. The drug is also contraindicated for patients with chronic intestinal diseases that alter digestion or absorption. Acarbose is contraindicated during pregnancy or breast-feeding. Patients with kidney dysfunction, DKA, or a hypersensitivity to the drug also shouldn’t take it.

Adverse Effects and Interactions

The most common reactions to acarbose are GI signs and symptoms. Patients may complain of abdominal pain, diarrhea, and flatulence caused by undigested carbohydrates in the intestines. These signs and symptoms tend to subside with time. At doses of 100 mg, acarbose may cause an asymptomatic, reversible increase in serum transaminase levels, especially in women.

The action of acarbose decreases with the concurrent use of charcoal, an intestinal adsorbent, and digestive enzyme preparations that contain carbohydrate-splitting enzymes, such as amylase and pancreatin. Hyperglycemia may result if a patient takes acarbose with thiazide and other diuretic drugs, beta-blockers, corticosteroids, phenothiazines, thyroid preparations, estrogens, oral contraceptives, phenytoin, nicotinic acid, sympathomimetics, calcium channel blockers, or isoniazid. Acarbose can be taken with a sulfonylurea or insulin, but these combinations may cause hypoglycemia. However, acarbose can help to prevent weight gain associated with sulfonylurea therapy.

Advise your patients to take acarbose with meals. Because the goal is to prevent the absorption of intestinal glucose, they should take the drug along with the first bite of food. No food interactions occur.

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Tests to Diagnose Diabetes

Blood Tests

Blood tests used to diagnose diabetes include the fasting blood glucose test, the 2-hour oral glucose tolerance test, and the random blood glucose test.

The glycosylated hemoglobin test is used to monitor the effectiveness of a patient’s therapy.

Tests to Diagnose Diabetes

Blood tests used to detect and monitor the progression of diabetic complications include a fasting lipid profile for cardiovascular disease and serum creatinine and blood urea nitrogen (BUN) tests for renal disease.

Diagnostic Tests

A physician may order diagnostic tests to diagnose diabetes, detect diabetic complications, or monitor the effectiveness of a patient’s therapy. Of course, a patient should also perform self-monitoring to check the effectiveness of her therapy.

Early detection of diabetic complications allows for early treatment, which can minimize their destructive effects. Tests that help diagnose a complication can be repeated to monitor its progress

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